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Indeed, loss-of-function Kelch-like ECH-associated protein 1 (KEAP1) mutations and gain-of-function nuclear factor erythroid 2-related factor 2 (NRF2) mutations lead to overactive NRF2 signalling in lung cancer relative to other tumour types

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This vulnerability forms the basis of the Phosphatidylcholine–Ferroptosis Axis, a key regulatory pathway linking lipid metabolism to ferroptotic cell death