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The Role of Brain-Derived Neurotrophic Factor as an Essential Mediator in Neuronal Functions and the Therapeutic Potential of Its Mimetics for Neuroprotection in Neurologic and Psychiatric Disorders - PMC

It caused HO in tendon tissue via activation of the mTOR pathway and suppression of autophagy both in TDSCs in vitro and in the Achilles tenotomy rat model [52]

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From the enzyme activities measured (i.e., CarE, CYP, AChE, and GST), GSTs showed the highest activity in response to berberine at both 48 h and compared with the other treatments

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