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Recently, ACMSD inhibitors were investigated in the human liver organoid model of metabolic dysfunction-associated steatohepatitis (MASH), an advanced form of liver fibrosis characterized by mitochondrial dysfunction

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Type 1 5α-reductase is expressed in the liver, kidney, skin, and brain, whereas type 2 5α-reductase is characteristically expressed strongly in the prostate but also at lower levels in the skin (hair follicles) and liver (125)