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Persistence of Bb infection in mice requires RpoS and involves RpoS-mediated repression of tick-phase genes Using a Δ rpoS strain complemented in trans (11), we previously demonstrated that loss of the complementing plasmid placed spirochetes at a survival disadvantage for up to 20 weeks following needle inoculation, supporting a requirement for RpoS during persistent infection

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Synaptic interaction of serotonergic axons and corticotropin releasing factor (CRF) synthesizing neurons in the hypothalamic paraventricular nucleus of the rat