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The flow of electrons from cysteine thiols can be directly shuttled to molecular oxygen by the action of the flavin-dependent family of sulfhydryl oxidases, such as Ero1p and Erv1, giving rise to a de novo disulfide bond formation [13, 14]

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These findings indicate that, unlike TLR4⁻ILC2s, adaptive immunity plays a limited role in modulating TLR4 + ILC2s