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There are other, perhaps more common, genetic variants associated with the risk of developing AD—including the late onset form of the disease (LOAD)—such as ApoE4 [521, 522] or those identified more recently (e.g., mutations in CD36 [523], see [524] for review)

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Insulin resistance is characterized by inadequate glucose uptake with altered glucose metabolism in peripheral tissues at a given concentration of plasma insulin (2)