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It upregulates the expression of anti-inflammatory genes, including NF-κBIA , MAP3K8 , and TLR5 , while concurrently downregulating pro-inflammatory genes such as TNFSF6 , TNF-α , and nuclear factor-κB ( NF-κB ) [36–39]

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The World Health Organization predicts it will be the third leading cause of death by 2030