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[DOI] [PMC free article] [PubMed] [Google Scholar] 24.Strbe S., Smoday I.M., Krezic I., Kalogjera L., Vukovic V., Zizek H., Gojkovic S., Vranes H., Barisic I., Sikiric S., et al

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This absence of a distinct phenotype is explained by the presence of the cytosolic NADPH-dependent Thioredoxin Reductase (NTR)/thioredoxin (TRX) system which serves as an alternative backup system for the reduction of cytosolic GSSG, albeit with a lower efficiency than GR1 [117]