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This dysfunction can be triggered by intrinsic host factors, such as autoimmune diseases (13–15), as well as extrinsic environmental factors, such as maternal infection and environmental exposures to infections during early life, childhood, and around the first episode of psychosis (16, 17)

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This process has been demonstrated to emulate the behavior of SOD and CAT, thereby scavenging free radicals and combating chronic inflammation and oxidative stress