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Favism and hemolytic anemia in glucose-6-phosphate dehydrogenase-deficient subjects in North Sardinia

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Reduced protein synthesis rates in HSCs with MYSM1 loss To begin to define how MYSM1 loss can compromise human HSC function, we examined the scRNA-seq data for those cells that had the molecular HSC signature and found that there was a significant downregulation of gene sets and genes implicated in ribosomal biogenesis and translation regulation (Figure 2A–C)