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This shift results in inefficient energy production and increased oxidative stress (56)

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A study using CXCR3 depletion antibodies to treat vitiligo mice with more than 75% depigmentation showed that CXCR3 depletion significantly reversed the reduction of pre-melanosomal protein in the epidermis and decreased CD8 + T cell numbers, resulting in significant repigmentation at the lesion site (Richmond et al., 2017)