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Another tryptophan-derived metabolite, indole-3-acetic acid (IAA), inhibits the TLR4/MyD88/NF-κB signaling pathway in M1 macrophages, promotes M2 macrophage polarization and restores the balance of M1/M2 polarization, thereby reducing aortic inflammation (83)

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Elevated levels of neutrophil activated proteins, alpha-defensins (DEFA1), calprotectin (S100A8/A9) and myeloperoxidase (MPO) are associated with disease severity in COVID-19 patients