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However, in Gilbert’s Syndrome the levels wouldn’t be high enough for long enough: In spite of the increased awareness of UCB effects to brain cells ( Brites and Brito, 2012 ), knowledge about its passage across the endothelial monolayer, the rate in which this passage is achieved, and its effects on the integrity of the brain endothelial barrier is scarce … In conclusion, our data demonstrate that UCB impairs barrier function in an in vitro model of the human BBB

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“Those two compounds alone have never been shown to have any benefit to justify them being used therapeutically in humans,” says Taylor

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