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Therapeutic strategies targeting cuproptosis in KOA Copper chelators, such as TTM and D-penicillamine, effectively sequester intracellular excess copper, thereby attenuating copper overload, suppressing FDX1-mediated cuproptosis, and preserving chondrocyte viability

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When arginine serves as the counterion for penta deca peptide arginate, it does more than simply stabilize the molecule