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This GR is also known to interact with other transcription factors like AP-1 and NF-kB, where the GR exerts an inhibitory effect on NF-kB, resulting in downregulation of proinflammatory genes under normal physiological conditions (130)

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TNF-alpha and IL-1 alpha inhibit both pyruvate dehydrogenase activity and mitochondrial function in cardiomyocytes: evidence for primary impairment of mitochondrial function