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Emerging evidence indicates that immune modulation is a strategic frontier: preclinical models demonstrate that NOD-like receptor protein 3 (NLRP3) inflammasome inhibitors decrease Interleukin-1β (IL-1β) levels and mitigate podocyte injury (21), Targeting monocyte chemoattractants with chemokine receptor 2 (CCR2) antagonists results in a significant reduction in urinary excretion of monocyte chemoattractant protein 1 (MCP-1) (22)
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