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In the same way, the depletion of LRRK2 or application of LRRK2 inhibitors decreases ROS, restores mitochondrial function, prevents mitochondrial fragmentation, and blocks increases in proapoptotic proteins including caspase 3, Bax, and apoptotic-inducing factor [53,54,55]

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Metabolic research leverages IGF-1 LR3’s effects on glucose uptake and insulin sensitivity