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In particular, peri-implant tissues lack a periodontal ligament and exhibit differences in vascularization and structural organization, which may influence disease progression and host response ( Among the biological processes implicated in periodontal and peri-implant tissue destruction, oxidative stress has emerged as a central and integrative pathogenic mechanism ( Within the periodontal microenvironment, neutrophils, macrophages, and activated fibroblasts generate high levels of superoxide anions, hydrogen peroxide, and hydroxyl radicals as part of the innate immune response