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However, a recent study conducted by DeNicola discovered that the glutamate-cysteine ligase catalytic subunit (GCLC) can defend against ferroptosis induced by cysteine deprivation through facilitating the formation of γ-glutamyl-peptides, which can be enhanced by NRF2 [66]

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This process allows its metabolites to act locally in the bladder, reducing inflammation and oxidative damage