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Human macrophages polarized by interaction with apoptotic cells produce fibrosis-associated mediators and enhance pro-fibrotic activity of dermal fibroblasts in vitro

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During SARS-CoV-2 infection, intense and rapid stimulation of the immune system response can trigger the activation of the NLRP3 inflammasome pathway and the release of its products, including IL-18 and IL-1β (Freeman and Swartz, 2020