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Preclinical studies found that treating neuronal, nematode, or rodent AD models with NR resulted in induction of a mitochondrial stress response, including UPR mt and mitophagy, reduced proteotoxic stress, and decreased Aβ accumulation 243

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(2009) more recently evaluated the contribution of human CBS-catalyzed reactions to cystathionine and H 2 S production by performing kinetic simulations at assumed physiological concentrations of serine, Cys, and Hcy (0.56, 0.10, and 0.01 mM, respectively)