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To further validate the decrease in cell viability was due to selective disruption of the FAK-paxillin interaction and not non-specific membrane damage, LDH assays were performed, and no significant levels of cytotoxicity were observed (Supplementary Fig

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TSG‐6 inhibits IL‐1beta‐induced inflammatory responses and extracellular matrix degradation in nucleus pulposus cells by activating the PI3K/Akt signaling pathway