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In DN, high glucose upregulates PRMT1, which mediates H4R3me2a modification at the activating transcription factor 6 (ATF6) promoter, activating the protein kinase R-like endoplasmic reticulum kinase (PERK) and ATF6 pathways and triggering the endoplasmic reticulum (ER) stress and apoptosis in tubular cells (Chen et al., 2019)